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枯萎病对温室黄瓜根际细菌生理菌群及酶活性的影响

余文英 詹光良 张绍升

福建农业学报2011,Vol.26Issue(4):605-610,6.
福建农业学报2011,Vol.26Issue(4):605-610,6.

枯萎病对温室黄瓜根际细菌生理菌群及酶活性的影响

Impact of Cucumber Fusarium wilt in Rhizosphere Microbe Flora and Enzyme Activity in Greenhouse

余文英 1詹光良 1张绍升2

作者信息

  • 1. 福建农林大学生命科学学院,福建福州 350002
  • 2. 福建农林大学植物保护学院,福建福州350002
  • 折叠

摘要

Abstract

By infection with Fusarium oxysporum in greenhouse, bacterial physiological flora of cucumber rhizosphere was changed and enzymatic activities decreased. The population of nitrobacteria and nitrite bacteria decreased in the early and peak phases of infection, thereafter which enhanced growing of aerobic azotobacter and denitrifier. The population of ammonifying bacteria and cellulose-decomposition bacteria consistently decreased during infection period. The population of rhizosphere thiobacillus dropped dramatically concomitant of increase in anti-sulfuration bacteria. The impact of nitrobacteria in cucumbers was significantly different between susceptible and resistant varieties, which may play an important role in physiologic accommodations against of cucumber Fusarium wilt. After Fusarium oxysporum infection, the activities of rhizosphere enzymes were declined significantly in catelase, urease, cellulase and alkaline-phosphatase. In early infection, the decline of activities was observed for catelase and cellulase more remarkable in susceptible varieties than in resistant varieties. However, the extent of decline in late of infection was found for urease and phosphatase but more remarkable in resistant varieties than that in susceptible. The impact of catelase in resistance to the disease was different significantly between susceptible and resistant varieties, which can be used as reference in resistant breeding.

关键词

温室/黄瓜枯萎病/根际/生理菌群/酶活性

Key words

greenhouse/ cucumber wilt/ rhizosphere/ Physiological microbe flora/ enzymatic activity

分类

农业科技

引用本文复制引用

余文英,詹光良,张绍升..枯萎病对温室黄瓜根际细菌生理菌群及酶活性的影响[J].福建农业学报,2011,26(4):605-610,6.

基金项目

福建省教育厅项目(2005K038) (2005K038)

福建农业学报

OACSTPCD

1008-0384

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