广东药学院学报2012，Vol.28Issue(6)：689-692,4.DOI:10.3969/j.issn.1006-8783.2012.06.026
β淀粉样蛋白神经毒性与线粒体功能异常研究进展
Research progress on neurotoxicity effect of β-amyloid protein and mitochondrial dysfunction
摘要
Abstract
Alzheimer's disease ( AD) is the most common type of Alzheimer s, and its pathogenesis is complex, p-amyloid protein ( AD) is considered to be the key factor of inducing AD. The neurotoxicity of Aβ mediates nerve cell mitochondrial dysfunction, which is an important event in AD pathogenesis. In the present paper,the neurotoxicity of Aβ and mitochondrial dysfunction are reviewed in recent years.关键词
β淀粉样蛋白/线粒体/阿尔茨海默病Key words
β-amyloid/mitochondria/Alzheimer' s disease分类
生物科学引用本文复制引用
赵美顺,陈敬荣,杨红..β淀粉样蛋白神经毒性与线粒体功能异常研究进展[J].广东药学院学报,2012,28(6):689-692,4.基金项目
广东省科技计划项目(2012B031800430 （）
2010B020312016) （）
