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脑梗死缺血再灌注损伤机制的研究进展

陈念 王立平

中国社区医师Issue(12):18-19,2.
中国社区医师Issue(12):18-19,2.DOI:10.3969/j.issn.1007-614x.2014.12.8

脑梗死缺血再灌注损伤机制的研究进展

Research progress of the mechanism of cerebral ischemic reperfusion injury

陈念 1王立平1

作者信息

  • 1. 301500天津市宁河县医院神经内科
  • 折叠

摘要

Abstract

Cerebral ischemia reperfusion injury is a research hotspot of acute cerebral infarction at present,and its mechanism is complex.In recent years,the study found that cerebral ischemia reperfusion injury has relationship with Na +-K +-ATP enzyme, glutathione peroxidase,nitric oxide synthas,peroxisome proliferators activated receptor gamma,Caspase-3,aquapon-4 and so on. Through this research,we can give intervention measures as soon as possible,reduce the degree of reperfusion injury after cerebral infarction,save more brain death,improve the ability of daily life of the patients.

关键词

脑梗死/脑缺血再灌注/损伤机制/研究进展

Key words

Cerebral infarction/Cerebral ischemic reperfusion/Injury mechanism/Research progress

引用本文复制引用

陈念,王立平..脑梗死缺血再灌注损伤机制的研究进展[J].中国社区医师,2014,(12):18-19,2.

中国社区医师

1007-614X

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