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The E3 ubiquitin ligase seven in absentia homolog 1 may be a potential new therapeutic target for Parkinson’s disease

Zeng-lin Cai Jing Xu Shou-ru Xue Yuan-yuan Liu Yong-jin Zhang Xin-zhi Zhang Xuan Wang Fang-ping Wu Xiao-min Li

中国神经再生研究(英文版)Issue(8):1286-1291,6.
中国神经再生研究(英文版)Issue(8):1286-1291,6.DOI:10.4103/1673-5374.162763

The E3 ubiquitin ligase seven in absentia homolog 1 may be a potential new therapeutic target for Parkinson’s disease

The E3 ubiquitin ligase seven in absentia homolog 1 may be a potential new therapeutic target for Parkinson’s disease

Zeng-lin Cai 1Jing Xu 1Shou-ru Xue 2Yuan-yuan Liu 2Yong-jin Zhang 1Xin-zhi Zhang 1Xuan Wang 1Fang-ping Wu 1Xiao-min Li1

作者信息

  • 1. Department of Neurology, Afifliated Lianyungang Hospital of Xuzhou Medical College, Lianyungang, Jiangsu Province, China
  • 2. Department of Neurology, First Afifliated Hospital of Soochow University, Suzhou, Jiangsu Province, China
  • 折叠

摘要

关键词

nerve regeneration/neurodegeneration/Parkinson’s disease/ubiquitin-proteasome system/autophagy/E3 ubiquitin ligase seven in absentia homolog 1/1-methyl-4-phenylpyridinium/rapa-mycin/neural regeneration

Key words

nerve regeneration/neurodegeneration/Parkinson’s disease/ubiquitin-proteasome system/autophagy/E3 ubiquitin ligase seven in absentia homolog 1/1-methyl-4-phenylpyridinium/rapa-mycin/neural regeneration

引用本文复制引用

Zeng-lin Cai,Jing Xu,Shou-ru Xue,Yuan-yuan Liu,Yong-jin Zhang,Xin-zhi Zhang,Xuan Wang,Fang-ping Wu,Xiao-min Li..The E3 ubiquitin ligase seven in absentia homolog 1 may be a potential new therapeutic target for Parkinson’s disease[J].中国神经再生研究(英文版),2015,(8):1286-1291,6.

基金项目

This study was supported by the China Postdoctoral Science Foundation, No.1630 ()

the Natural Science Foundation of Jiangsu Province in China, No. BK2011402 ()

the Jiangsu Province Postdoctoral Research Foundation in China, No.1301174C ()

the Jiangsu Province Health Depart-ment Foundation in China, No. H201361 ()

中国神经再生研究(英文版)

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1673-5374

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