中华神经创伤外科电子杂志2016，Vol.2Issue(3)：173-177,5.DOI:10.3877/cma.j.issn.2095-9141.2016.03.013
创伤性脑损伤后凝血功能障碍的发生机制和治疗进展
Pathogenesis and treatment progress of coagulation dysfunction after traumatic brain injury
摘要
Abstract
There is a close relationship between the traumatic brain injury and the functional changes of coagulation system. However, there is still a lack of in-depth study on the mechanism of the interaction between the two. The high coagulation state and low coagulation state after traumatic brain injury can lead to the formation of micro thrombosis or aggravate intracranial hemorrhage, which can lead to a series of secondary injury. Therefore, researchers have proposed several hypotheses, such as tissue factor release, disseminated intravascular coagulation, fibrinolysis and platelet dysfunction etc. This article reviews the pathogenesis, clinical diagnosis, treatment and prognosis of the abnormal coagulation function after traumatic brain injury.关键词
创伤性脑损伤/创伤后凝血功能障碍/组织因子Key words
Traumatic brain injury/Arauma-induced coagulopathy/Tissue factor引用本文复制引用
张全,田恒力..创伤性脑损伤后凝血功能障碍的发生机制和治疗进展[J].中华神经创伤外科电子杂志,2016,2(3):173-177,5.基金项目
国家自然科学基金资助项目（81271383、81471245）；上海市科技基金资助项目 （）
