中华神经创伤外科电子杂志2016，Vol.2Issue(5)：302-304,3.DOI:10.3877/cma.j.issn.2095-9141.2016.05.010
创伤性颅脑损伤继发凝血功能异常的发生机制
Research progress of mechanism about the secondary coagulation dysfunction following the traumatic brain injury
摘要
Abstract
Coagulant abnormality can occur secondary to traumatic brain injury (TBI). The mechanism includes platelet dysfunction,abnormal activation of UPA/TPA, brain-derived microparticulars (BDMP), activation of protein C system, release of tissue factors (TF), change of blood coagulation factor Ⅶ activity, etc. In this review, recently researches were briefly reviewed to illuminate the possible mechanisms of this secondary coagulant abnormality in the different phases of TBI.关键词
创伤性颅脑损伤/凝血功能异常/研究进展Key words
Traumatic brain injury/Coagulation abnormalities/Research progress引用本文复制引用
李田飞,李军,魏梁锋,王守森..创伤性颅脑损伤继发凝血功能异常的发生机制[J].中华神经创伤外科电子杂志,2016,2(5):302-304,3.基金项目
全军后勤科技重大项目 （）
