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脑出血后脑水肿的形成机制及药物治疗研究进展

韩鹏 李力仙

临床与病理杂志2018,Vol.38Issue(2):424-428,5.
临床与病理杂志2018,Vol.38Issue(2):424-428,5.DOI:10.3978/j.issn.2095-6959.2018.02.031

脑出血后脑水肿的形成机制及药物治疗研究进展

Mechanism of brain edema after intracerebral hemorrhage and research progress of drug therapy

韩鹏 1李力仙1

作者信息

  • 1. 哈尔滨医科大学附属临床第一临床医院神经外科,哈尔滨 150001
  • 折叠

摘要

Abstract

Cerebral edema is an inevitable pathophysiological process after intracerebral hemorrhage. It is the most serious complication after intracerebral hemorrhage and leads to second brain injury in brain tissue after hemorrhage. Cerebral edema has serious adverse consequences. Increased extracellular fluid leads to increased intracranial pressure, resulting in impaired neurological function. The pathogenesis of cerebral edema is more and more complex; only based on pathogenesis and risk factors for symptomatic treatment and elimination of intracranial hypertension are feasible methods to treat edema. It is necessary to develop new and effective anti-edema drugs.

关键词

脑出血/脑水肿/病理机制/药物治疗

Key words

intracerebral hemorrhage/brain edema/mechanism/drug therapy

引用本文复制引用

韩鹏,李力仙..脑出血后脑水肿的形成机制及药物治疗研究进展[J].临床与病理杂志,2018,38(2):424-428,5.

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