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The role of sequestosome 1/p62 protein in amyotrophic lateral sclerosis and frontotemporal dementia pathogenesis

Adriana Delice Foster Sarah Lyn Rea

中国神经再生研究（英文版）2020，Vol.15Issue(12)：2186-2194,9.

中国神经再生研究（英文版）2020，Vol.15Issue(12)：2186-2194,9.DOI:10.4103/1673-5374.284977

The role of sequestosome 1/p62 protein in amyotrophic lateral sclerosis and frontotemporal dementia pathogenesis

Adriana Delice Foster ¹Sarah Lyn Rea²

作者信息

1. Harry Perkins Institute of Medical Research,University of Western Australia
2. Perron Institute for Neurological and Translational Science,Centre for Neuromuscular and Neurological Disorders,The University of Western Australia,Nedlands,Western Australia,Australia
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摘要

关键词

aggregate/inclusion body formation/amyotrophic lateral sclerosis-frontotemporal lobar degeneration/autophagy/cell signaling/mitophagy/p62/SQSTM1/protein degradation

Key words

aggregate/inclusion body formation/amyotrophic lateral sclerosis-frontotemporal lobar degeneration/autophagy/cell signaling/mitophagy/p62/SQSTM1/protein degradation

引用本文复制引用

Adriana Delice Foster,Sarah Lyn Rea..The role of sequestosome 1/p62 protein in amyotrophic lateral sclerosis and frontotemporal dementia pathogenesis[J].中国神经再生研究（英文版）,2020,15(12):2186-2194,9.

基金项目

This work was supported by the NHMRC-ARC Dementia Research Development Fellowship Grant (AP1102977) and an Australian Government Research Training Program (RTS) Scholarship. （AP1102977）

中国神经再生研究（英文版）

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ISSN：1673-5374

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