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Alcohol-induced Wnt signaling inhibition during bone fracture healing is normalized by intermittent parathyroid hormone treatment

Esha M.Kapania Taylor J.Reif Aaron Tsumura Jonathan M.Eby John J.Callaci

动物模型与实验医学(英文)2020,Vol.3Issue(2):200-207,8.
动物模型与实验医学(英文)2020,Vol.3Issue(2):200-207,8.DOI:10.1002/ame2.12116

Alcohol-induced Wnt signaling inhibition during bone fracture healing is normalized by intermittent parathyroid hormone treatment

Alcohol-induced Wnt signaling inhibition during bone fracture healing is normalized by intermittent parathyroid hormone treatment

Esha M.Kapania 1Taylor J.Reif 2Aaron Tsumura 3Jonathan M.Eby 3John J.Callaci4

作者信息

  • 1. Internal Medicine-Pediatric Resident,Rush University Medical Center,Chicago,IL,USA
  • 2. Limb Lengthening and Complex Reconstruction Fellowship,Hospital for Special Surgery,New York,NY,USA
  • 3. Department of Orthopaedic Surgery and Rehabilitation,Loyola University Medical Center,Maywood,IL,USA
  • 4. Alcohol Research Program (ARP),Loyola University Chicago Stritch School of Medicine,Maywood,IL,USA
  • 折叠

摘要

关键词

ethanol/fracture callus/parathyroid hormone/Wnt signaling pathway

Key words

ethanol/fracture callus/parathyroid hormone/Wnt signaling pathway

引用本文复制引用

Esha M.Kapania,Taylor J.Reif,Aaron Tsumura,Jonathan M.Eby,John J.Callaci..Alcohol-induced Wnt signaling inhibition during bone fracture healing is normalized by intermittent parathyroid hormone treatment[J].动物模型与实验医学(英文),2020,3(2):200-207,8.

基金项目

AO North America ()

National Institute on Alcohol Abuse and lcoholism, Grant/Award Number: R21AA025551 and T32AA013527 ()

动物模型与实验医学(英文)

OACSCD

2096-5451

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