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Altered O-GlcNAcylation and mitochondrial dysfunction, a molecular link between brain glucose dysregulation and sporadic Alzheimer 's disease

Chia-Wei Huang Nicholas C.Rust Hsueh-Fu Wu Gerald W.Hart

中国神经再生研究（英文版）2023，Vol.18Issue(4)：779-783,5.

Altered O-GlcNAcylation and mitochondrial dysfunction, a molecular link between brain glucose dysregulation and sporadic Alzheimer 's disease

Chia-Wei Huang ¹Nicholas C.Rust ²Hsueh-Fu Wu ¹Gerald W.Hart²

作者信息

1. Complex Carbohydrate Research Center,University of Georgia,Athens,GA,USA
2. Department of Biochemistry and Molecular Biology,University of Georgia,Athens,GA,USA
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摘要

关键词

Alzheimer's disease/amyloid beta/brain/glucose deficiency/glucose uptake/hypometabolism/mitochondrial dysfunction/neurodegenerative disease/neurons/O-GlcNAc/Tau

Key words

Alzheimer's disease/amyloid beta/brain/glucose deficiency/glucose uptake/hypometabolism/mitochondrial dysfunction/neurodegenerative disease/neurons/O-GlcNAc/Tau

引用本文复制引用

Chia-Wei Huang,Nicholas C.Rust,Hsueh-Fu Wu,Gerald W.Hart..Altered O-GlcNAcylation and mitochondrial dysfunction, a molecular link between brain glucose dysregulation and sporadic Alzheimer 's disease[J].中国神经再生研究（英文版）,2023,18(4):779-783,5.

基金项目

This work was supported by the Georgia Research Alliance and the University of Georgia(to GWH). （to GWH）

中国神经再生研究（英文版）

OACSCDCSTPCD

ISSN：1673-5374

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