Zoological Research2024，Vol.45Issue(4)：P.845-856,12.DOI:10.24272/j.issn.2095-8137.2023.363
SIL1 improves cognitive impairment in APP23/PS45 mice by regulating amyloid precursor protein processing and Aβ generation
Qunxian Wang 1Yanshuang Jiang 1Zijun Meng 1Xiangjun Dong 1Dongjie Hu 1Liangye Ji 1Weihui Zhou 1Weihong Song2
作者信息
- 1. Chongqing Key Laboratory of Child Neurodevelopment and Cognitive Disorders,Ministry of Education Key Laboratory of Child Development and Disorders,National Clinical Research Center for Child Health and Disorders,China International Science and Technology Cooperation Base of Child Development and Critical Disorders,Children''s Hospital of Chongqing Medical University,Chongqing 400000,China
- 2. Chongqing Key Laboratory of Child Neurodevelopment and Cognitive Disorders,Ministry of Education Key Laboratory of Child Development and Disorders,National Clinical Research Center for Child Health and Disorders,China International Science and Technology Cooperation Base of Child Development and Critical Disorders,Children''s Hospital of Chongqing Medical University,Chongqing 400000,China Second Affiliated Hospital and Yuying Children’s Hospital,Institute of Aging,Key Laboratory of Alzheimer’s Disease of Zhejiang Province,Wenzhou Medical University,Wenzhou,Zhejiang 325000,China Oujiang Laboratory(Zhejiang Lab for Regenerative Medicine,Vision and Brain Health),Wenzhou,Zhejiang 325001,China
- 折叠
摘要
关键词
Alzheimer’s disease/SIL1/APP processing/Cognitive impairment分类
医药卫生引用本文复制引用
Qunxian Wang,Yanshuang Jiang,Zijun Meng,Xiangjun Dong,Dongjie Hu,Liangye Ji,Weihui Zhou,Weihong Song..SIL1 improves cognitive impairment in APP23/PS45 mice by regulating amyloid precursor protein processing and Aβ generation[J].Zoological Research,2024,45(4):P.845-856,12.基金项目
National Natural Science Foundation of China(82230043,82293642)。 （82230043,82293642）
