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AKR1C3 protects cardiomyocytes against hypoxia-induced cell apoptosis through the Nrf-2/NF-κB pathway

Wenlu Zhang Wei Tian Xin Xia Hua Tian Ting Sun

Acta Biochimica et Biophysica Sinica2025，Vol.57Issue(7)：P.1151-1163,13.

Acta Biochimica et Biophysica Sinica2025，Vol.57Issue(7)：P.1151-1163,13.DOI:10.3724/abbs.2024230

AKR1C3 protects cardiomyocytes against hypoxia-induced cell apoptosis through the Nrf-2/NF-κB pathway

Wenlu Zhang ¹Wei Tian ²Xin Xia ¹Hua Tian ³Ting Sun¹

作者信息

1. Department of Cardiology,Shanghai Ninth People’s Hospital,Shanghai Jiao Tong University School of Medicine,Shanghai 200025,China
2. State Key Laboratory of Systems Medicine for Cancer,Shanghai Cancer Institute,Renji Hospital,Shanghai Jiao Tong University School of Medicine,Shanghai 20032,China
3. State Key Laboratory of Systems Medicine for Cancer,Shanghai Cancer Institute,Renji Hospital,Shanghai Jiao Tong University School of Medicine,Shanghai 20032,China Department of Pathology,the Affiliated Hospital of Youjiang Medical University for Nationalities,Baise 533000,China Key Laboratory of Molecular Pathology in Tumors of Guangxi Higher Education Institutions,Baise 533000,China
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摘要

关键词

AKR1C3/hypoxia/cardiomyocyte apoptosis/Nrf-2/NF-κB

分类

医药卫生

引用本文复制引用

Wenlu Zhang,Wei Tian,Xin Xia,Hua Tian,Ting Sun..AKR1C3 protects cardiomyocytes against hypoxia-induced cell apoptosis through the Nrf-2/NF-κB pathway[J].Acta Biochimica et Biophysica Sinica,2025,57(7):P.1151-1163,13.

基金项目

supported by the grant from the National Natural Science Foundation of China(No.81770505). （No.81770505）

Acta Biochimica et Biophysica Sinica

ISSN：1672-9145

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