首页|期刊导航|国际肝胆胰疾病杂志(英文版)|Chemokines and PI3K/AKT signaling pathway mediate the spontaneously ruptured hepatocellular carcinoma through the regulation of the cell cycle
国际肝胆胰疾病杂志(英文版)2025,Vol.24Issue(5):511-526,16.DOI:10.1016/j.hbpd.2025.01.003
Chemokines and PI3K/AKT signaling pathway mediate the spontaneously ruptured hepatocellular carcinoma through the regulation of the cell cycle
Chemokines and PI3K/AKT signaling pathway mediate the spontaneously ruptured hepatocellular carcinoma through the regulation of the cell cycle
摘要
关键词
Spontaneously ruptured hepatocellular carcinoma/Non-ruptured hepatocellular carcinoma/Transcriptome/Cell cycle/ChemokinesKey words
Spontaneously ruptured hepatocellular carcinoma/Non-ruptured hepatocellular carcinoma/Transcriptome/Cell cycle/Chemokines引用本文复制引用
Yan Zhang,Yang-Liu Zhou,Na Xu,Tao Meng,Zhen-Zhen Wang,Fa-Ming Pan,Li-Xin Zhu..Chemokines and PI3K/AKT signaling pathway mediate the spontaneously ruptured hepatocellular carcinoma through the regulation of the cell cycle[J].国际肝胆胰疾病杂志(英文版),2025,24(5):511-526,16.基金项目
This research was supported by grants from the National Natu-ral Science Foundation of China(52072005 and 51872279). (52072005 and 51872279)