农药学学报2026，Vol.28Issue(1)：161-169,9.DOI:10.16801/j.issn.1008-7303.2025.0102
草甘膦胁迫对茶树生理生化及次生代谢产物的影响
Effects of glyphosate stress on physiological,biochemical and secondary metabolites of tea plant
摘要
Abstract
To explore the effects of glyphosate stress on the physiological metabolism of tea plants,the changes in leaf ultrastructure,physiological and biochemical parameters,antioxidant system,and metabolite profiles were systematically investigated under simulated glyphosate exposure at varying concentrations.The results showed that glyphosate stress induced cellular structural damage.As the glyphosate concentration increased,the chlorophyll content declined.The activities of superoxide dismutase(SOD),catalase(CAT),peroxidase(POD),and glutathione peroxidase(GSH-Px),along with malondialdehyde(MDA)content,generally exhibited a trend of initial increase followed by a decline.The contents of free amino acids,epigallocatechin(EGC),epigallocatechin gallate(EGCG),epicatechin(EC),and epicatechin gallate(ECG)gradually decreased.Similarly,the total content of eight flavonoids showed a decreasing trend.In contrast,the contents of protocatechuic acid,shikimic acid,and gallic acid increased with rising glyphosate levels.These findings showed that glyphosate impairs the antioxidant defense system of tea plants,triggering a defensive response to reactive oxygen species(ROS)accumulation under stress.Furthermore,glyphosate affected tea plant metabolism by inhibiting the key enzymatic activities and reducing metabolic precursors,thereby inhibiting the biosynthesis of secondary metabolites.This study provides a theoretical basis for the rational application of glyphosate in tea gardens and for the breeding of tea varieties with enhanced stress tolerance.关键词
茶树/草甘膦/生理生化响应/次生代谢产物Key words
tea plant/glyphosate/physiological response/secondary metabolites分类
农业科技引用本文复制引用
方灵,史梦竹,韦航,梁启富,傅建炜..草甘膦胁迫对茶树生理生化及次生代谢产物的影响[J].农药学学报,2026,28(1):161-169,9.基金项目
福建省属公益项目(NO.2022R1022001). Supported by Fujian Public Welfare Project(NO.2022R1022001). （NO.2022R1022001）
