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首页|期刊导航|吉林中医药|基于中医"肺热叶焦,则生痿躄"理论探讨慢阻肺与肌骨共病交互机制

基于中医"肺热叶焦,则生痿躄"理论探讨慢阻肺与肌骨共病交互机制

王海强 王彬 金子开 郑力铭 高峰 魏戌

吉林中医药2026,Vol.46Issue(8):969-974,6.
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吉林中医药2026,Vol.46Issue(8):969-974,6.DOI:10.13463/j.cnki.jlzyy.2026.08.005

基于中医"肺热叶焦,则生痿躄"理论探讨慢阻肺与肌骨共病交互机制

Exploration of the interaction mechanism of COPD and musculoskeletal comorbidities based on the"scorched lung causing atrophy and flaccidity"theory in traditional Chinese medicine

王海强 1王彬 1金子开 1郑力铭 1高峰 1魏戌2

作者信息

  • 1. 中国中医科学院望京医院,北京 100102
  • 2. 中国中医科学院望京医院,北京 100102||中国中医科学院望京医院南阳医院 南阳市中医院,河南 南阳 473003
  • 折叠

摘要

Abstract

Chronic obstructive pulmonary disease(COPD)is a lung disease characterized by high rates of morbidity,disability,and mortality,often accompanied by musculoskeletal diseases such as sarcopenia and osteoporosis(OP).Although the pathogeneses underlying COPD and musculoskeletal comorbidities are not yet fully unveiled,recent studies suggest that pulmonary,muscle,and bone factors communicate through cross-organ signaling,constituting a"lung-musculoskeletal interaction"mechanism that plays a key role in the onset and progression of these comorbidities.The theory of"scorched lung causing atrophy and flaccidity"from the Inner Canon of Huangdi provides a theoretical basis for the pathological relationship between the lung and musculoskeletal system diseases.This paper combines this theory with modern biomedical mechanisms to explore the interrelationship between COPD and musculoskeletal comorbidities,further elucidating the role of the"lung-musculoskeletal interaction"mechanism in the development of these comorbidities.

关键词

慢性阻塞性肺疾病/肌少症/骨质疏松症/肺-肌骨交互/肺热叶焦

Key words

chronic obstructive pulmonary disease/sarcopenia/osteoporosis/lung-musculoskeletal crosstalk/scorched lung

分类

医药卫生

引用本文复制引用

王海强,王彬,金子开,郑力铭,高峰,魏戌..基于中医"肺热叶焦,则生痿躄"理论探讨慢阻肺与肌骨共病交互机制[J].吉林中医药,2026,46(8):969-974,6.

基金项目

中国中医科学院望京医院创新团队项目(WJCXTD-202408) (WJCXTD-202408)

中国中医科学院卓越青年科技人才培养专项(CI2024D003) (CI2024D003)

中国中医科学院科技创新工程项目(CI2026A02028) (CI2026A02028)

吉林中医药

1003-5699

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