Temporally regulated FGFR2-retinoic acid signaling mediates dura mater-suture mesenchyme interactions to prevent craniosynostosis in mice
Temporally regulated FGFR2-retinoic acid signaling mediates dura mater-suture mesenchyme interactions to prevent craniosynostosis in mice
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Lu Gao,Peng Chen,Jifan Feng,Tingwei Guo,Mingyi Zhang,Thach-Vu Ho,Jian-Fu Chen,Yang Chai..Temporally regulated FGFR2-retinoic acid signaling mediates dura mater-suture mesenchyme interactions to prevent craniosynostosis in mice[J].国际口腔科学杂志（英文版）,2026,18(4):714-727,14.基金项目
We gratefully acknowledge Bridget Samuels,Kimi Nakaki,and Linda Hattemer(Center for Craniofacial Molecular Biology,USC)for their critical review of the manuscript.We wish to thank Ivetta Vorobyova(Molecular Imaging Center,USC)for all the live Micro CT scanning.We also wish to thank the USC Office of Research and the Norris Medical Library for their support in providing bioinformatics software and computing resources.Our sincere appreciation goes to Dr.Jinsook Son and Dr.Domenico Accili at Columbia University for sharing the Aldh1a3fl/+mouse strain used in this study.We are deeply grateful to Dr.Shaifur Rahman for his valuable contributions to the early stages of this project by conducting preliminary experiments that helped identify the initial craniosynostosis phenotype on the Gli1-CreERT2 （Center for Craniofacial Molecular Biology,USC）
Fgfr2IIIcfl/+mice.This work was funded by the National Institute of Dental and Craniofacial Research,National Institutes of Health(NIDCR,NIH R01DE030901 to J.C.and Y.C.). （NIDCR,NIH R01DE030901 to J.C.and Y.C.）
